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These forms of epigenetic modifications interact with each other to jointly regulate gene expression, playing critical roles in processes such as cell differentiation, development, disease progression, and stress responses [17, 18]

That was one of the things I was going to ask you about and comment on because it's always interesting when you're writing a book or having a project like this

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MITOL/MARCH5 determines the susceptibility of cardiomyocytes to doxorubicin-induced ferroptosis by regulating GSH homeostasis

How it fits with everything else I'm a functional medicine physician, so I don't think in terms of a single magic input