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In vitro experiments showed that CHE could inhibit the proliferation of OC cells A2780 and SKOV3 and induce ferroptosis, while combined treatment with the Nrf2 activator TBHQ could reverse the CHE-induced ferroptosis effect

Bordoni L, Gabbianelli R

Soc Dev 3(2):108124

doi: 10.7554/eLife.62852 168 ZhangD.ChenG.ManwaniD.MorthaA.XuC.FaithJ

Inflammation, oxidative stress, Ca 2+ dysregulation, and hypoxia can induce cellular endoplasmic reticulum stress (ERS) by overloading the ERs protein folding mechanism ( Some studies have found that ferroptosis and ERS share a common pathway, especially in tumor diseases, and disease trends can be altered by controlling the ERS or ferroptosis pathway ( 2 Ferroptosis in RA With the study of RA disease mechanisms, it has been found that the aberrant activation of RA-FLSs, destruction of articular cartilage, overactivation of immune cells, and sustained expression of inflammatory factors are associated with ferroptosis

Although various pathophysiological mechanisms and genetic factors have been proven to be related to NAFLD, its treatment remains challenging